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Rescuing BMPR2-driven endothelial dysfunction in PAH: a novel treatment strategy for the future?

  
@article{SCI15269,
	author = {Rozenn Quarck and Fr├ęderic Perros},
	title = {Rescuing BMPR2-driven endothelial dysfunction in PAH: a novel treatment strategy for the future?},
	journal = {Stem Cell Investigation},
	volume = {4},
	number = {6},
	year = {2017},
	keywords = {},
	abstract = {Pulmonary arterial hypertension (PAH) may be idiopathic and corresponds to sporadic disease without any familial history or identified risk factors, or heritable when it occurs in the hereditary context; germline mutations in the bone morphogenetic protein receptor type 2 (BMPR2) gene, are detected in 70% of heritable PAH cases (1). Even in sporadic cases, PAH is associated with impaired BMPRII signalling, occurring through small mothers against decapentaplegic (SMAD) proteins (2).},
	issn = {2306-9759},	url = {http://sci.amegroups.com/article/view/15269}
}